Consumer Alert: BPA-Free Goods Still Contain Toxic Bisphenol

Breaking research indicates that manufacturers throughout the world who were using the toxic hormone-disrupting chemical bisphenol A (BPA), may have simply switched to an equally toxic analogue in the same bisphenol chemical class known as bisphenol S (BPS), to evade regulatory oversight and trick consumers with their misleading “BPA-FREE!” labels….

If you are already sensitive to the highly concerning issue of toxic chemicals in everyday consumer products, you’ve probably noticed these “BPA-Free” labels popping up everywhere.  Bisphenol A (BPA), of course, is a chemical capable of disrupting our hormones, and is especially problematic in pregnancy, infancy and childhood — times of greatest susceptibility to the adverse effects of environmental exposures.

Now that consumers are refusing to buy BPA-containing containers, manufacturers have had no choice but to oblige with seemingly bisphenol-free alternatives. Ironically, BPA is used to make Sippy Cups (and polycarbonate) shatterproof, and therefore “safer.” But, instead of manufacturers actually removing the danger in BPA-free labeled products, many are capitalizing on this marketing opportunity by removing only the perception of danger, opting to substitute BPA for equally toxic members of the bisphenol chemical class.

An Alphabetic Soup of Toxic Bisphenols In Consumer Goods

While BPA has received the most negative attention, often being labeled as “bisphenol” for short, the bisphenols are such a broad chemical class that they are identified pseudo-alphabetically, spanning letters A-Z:

1. Bisphenol A


2. Bisphenol AB


3. Bisphenol AF


4. Bisphenol B


5. Bisphenol BP


6. Bisphenol C


7. Bisphenol E


8. Bisphenol F


9. Bisphenol G


10. Bisphenol M


11. Bisphenol S


12. Bisphenol P


13. Bisphenol PH


14. Bisphenol TMC


15. Bisphenol Z

Bisphenol A has become the primary focus of consumer advocates for several reasons:

1) It is the most extensively research bisphenol, with at least 40 potential adverse health effects identified in the biomedical literature thus far.

2) Canada became the first country to officially declare BPA a “toxic substance” in September 2010[1]

3) It has the highest production volume and use in consumer goods, with 2.2 million tons consumed globally in 2009.

Primary routes of exposure include:

1. Plastics


2. PVC water lines


3. Canned Goods


4. Thermal Receipt Paper


5. All World Paper Currency


6. Dental Sealants

Despite industry spokespersons and industry-funded scientists claiming BPA does not possess estrogenic properties of any biological significance, it was first acknowledged and used as an artificial estrogen in the early 1930’s, where it was used both to fatten poultry and cattle, as well as a form of estrogen replacement therapy in women in the mid-1930’s.[2]  Only later, in the 1940’s, did Bayer and General Electric use BPA to harden polycarbonate plastics and make epoxy resin, the latter of which is still used to line nearly the entire world’s supply of food and beverage containers.  Only Japanese industries, which responded to consumer concern about the toxic effects of BPA between 1998-2003, voluntarily reduced BPA in favor of safer alternatives.

In the United States and abroad, in response to growing consumer concern over the safety of BPA, manufacturers have been rapidly substituting Bisphenol S (and possibly other bisphenol analogues) in its place.

How do we know this? After all, manufacturers are not required to list bisphenols explicitly on the labels of the consumer products within which they are used. Two new studies reveal that Bisphenol S (BPS) has become as dominant as BPA in manufacturing paper products we come into contact daily, as well as in terms of measurable human exposure….

New Research Proves Bisphenol A Is Secretly Being Replaced By Bisphenol S

First, a study published last week in the journal Environmental Science & Technology found BPS in thermal paper from stores in the US, Japan, South Korea and Vietnam, at concentrations similar to earlier reports of BPA. Concentrations were found as high as 22 mg per gram of paper — two orders of magnitude higher concentrations than are considered biologically significant. BPS was also found in 87 percent of the paper currency assayed from 21 countries, indicating it is being utilized on a  truly global scale.

The same week the same journal published another study titled, “Bisphenol S in Urine from the United States and Seven Asian Countries: Occurrence and Human Exposures,”[3] revealing for the first time that Bisphenol S concentrations in urine from the citizens of 8 countries were within the same concentration ranges as Bisphenol A levels reported by the same research group in the year before.[4]

Is Bisphenol S Safer Because We Know Less About It?

Despite the industry-wide move away from BPA towards BPS, they both exhibit similar estrogen-like properties.  A 2005 study performed by a Japanese research group found that BPS was only a slightly weaker estrogen than BPA.  But, as the Environmental Health News reported in 2010:

They [Japanese researchers] tested the effects on human cells and found that bisphenol S was slightly less potent than BPA, but not by much: bisphenol S was active at 1.1 micromolar concentration, BPA at 0.63 micromolar. One micromolar is roughly equivalent to a packet of sugar in 3,000 gallons of water.[5]

A growing body of additional research now indicates that Bisphenol S is both estrogenic and uterotrophic (increasing the weight of the uterus), with clearly carcinogenic potential.[6] [7] [8] [9] [10]

A 2012 study published in the journal Toxicology In Vitro discussed the industry-wide shift from using BPA in plastic baby bottles to BPS after the European Commission imposed a restriction on BPA use in 2011, as a  direct result of scientific concerns over its estrogenic properties. The researchers found that “By using two highly standardised transactivation assays, we could demonstrate that the estrogenic activity of Bisphenol A and Bisphenol S is of a comparable potency.”

The problem is that BPS is less well-known and researched than BPA for its potential adverse effects, and while regulators wait for manufacturers who promote their products with “BPA-Free!” stickers at the same moment that they infuse them with BPS to voluntarily reformulate, there is evidence now that BPS may actually have worse effects to environmental and human health, alike..

One of the main concerns is that BPS is significantly less biodegradable than BPA. According to a study published in the International Journal of Environmental Research & Public Health in 2009, BPS “… is not amenable to biodegradation and might be persistent and become an ecological burden.”[11]  Another study published in the journal Environmental Toxicology & Pharmacology in 2005 found that BPS is “…more heat-stable and photo-resistant than bisphenol A.”[12]

BPS’ relative inability to biodegrade indicates: 1) once it is absorbed into the human body, it may accumulate there for longer periods of time. 2) it is more likely to persist in the environment, making external exposures to it, and its many metabolites, much more likely than the faster degrading BPA. In other words, its potential to do harm will worsen along the axis of time, not lessen, which is a common argument made for the purported “safety” of BPA.

Are We A Population of Guinea Pigs Without The Precautionary Principle?

Recently, it was discovered that synthetic estrogens are leaching into our bodies from consumer products (e.g. parabens) and are now eclipsing natural human estrogens.  A paraben ester was found at 1 million times higher concentrations than natural estrogen levels in the breast tissue of mastectomy patients. After several generations of “better living through chemistry,” we are discovering that we have poisoned ourselves and our environment (can we really separate them any longer?) into profound and possibly irrevocable disfigurement.  What is needed now more than ever is a paradigm shift in how we regulate our exposure to chemicals, and the industries that produce them virtually unrestricted.

Perhaps we must begin asking ourselves: What would the chemical and drug industries do without the twin disciplines of toxicology and pharmacology? The former provides ample justification for continually exposing us to “an acceptable level of harm” from the chemicals it secretes, and the latter convinces us just how we can’t live without “therapeutic” chemicals, e.g. pharmaceuticals.

Until we demand the implementation of the precautionary principle in this country, and abroad, nothing will change … other than, increasingly, our very biological and genetic integrity, for the worse.

---

[1] Canada Gazette Part II. 13 October 2010;144(21):1806–18.

[2] Erler, C. & Novak, J. Bisphenol A Exposure: Human Risk and Health Policy. Journal of Pediatric Nursing. 2010. Volume 25:400-407. Accessed online March 1, 2012.

[3] Bisphenol S in Urine from the United States and Seven Asian Countries: Occurrence and Human Exposures. Environ Sci Technol. 2012 May 23. Epub 2012 May 23. PMID: 22620267

[4] Bisphenol S in Urine from the United States and Seven Asian Countries: Occurrence and Human Exposures. Environ Sci Technol. 2012 May 23. Epub 2012 May 23. PMID: 22620267

[5] Environmental Health News, Nov. 12, 2010 BPA: What’s the alternative?

[6] Weak estrogenic transcriptional activities of Bisphenol A and Bisphenol S. Toxicol In Vitro. 2012 Aug ;26(5):727-31. Epub 2012 Apr 5. PMID: 22507746

[7] Comparative study of the uterotrophic potency of 14 chemicals in a uterotrophic assay and their receptor-binding affinity. Toxicol Lett. 2004 Jan 15 ;146(2):111-20. PMID: 14643963

[8] Acute toxicity, mutagenicity, and estrogenicity of bisphenol-A and other bisphenols. Environ Toxicol. 2002 Feb ;17(1):80-6. PMID: 11847978

[9] Estrogenic activity of dental materials and bisphenol-A related chemicals in vitro. Dent Mater J. 2000 Sep ;19(3):245-62. PMID: 11218845

[10] Measurement of estrogenic activity of chemicals for the development of new dental polymers. Toxicol In Vitro. 2001 Aug-Oct;15(4-5):421-5. PMID: 11566573

[11] Biodegradation of bisphenol A, bisphenol F and bisphenol S in seawater. Int J Environ Res Public Health. 2009 Apr ;6(4):1472-84. Epub 2009 Apr 17. PMID: 19440529

[12] Estrogenic activity of alkylphenols, bisphenol S, and their chlorinated derivatives using a GFP expression system. Environ Toxicol Pharmacol. 2005 Jan ;19(1):121-30. PMID: 21783468

© April 13, 2018 GreenMedInfo LLC. This work is reproduced and distributed with the permission of GreenMedInfo LLC. Want to learn more from GreenMedInfo? Sign up for the newsletter here http://www.greenmedinfo.com/greenmed/newsletter.

The post Consumer Alert: BPA-Free Goods Still Contain Toxic Bisphenol appeared first on The Sleuth Journal.

MSG: Delicious Seasoning Or Drug And Poison?

Poor food quality is a growing problem in industrialized countries increasingly focused on the quantitative dimension of food production, both in terms of the sheer volume of food produced, as well as the revenue and profit generated. In the United States, for instance, consumers do not even have the right to know what is in their food, e.g. genetically modified ingredients are not labeled by default; the use of paint pigment (titanium dioxide) as a “manufacturing aid” in milk, not required to be listed as an ingredient on the label, etc, and raw human sewage and factory-farmed animal waste, as well as petroleum and coal byproducts, are all considered fair game as a growing medium in USDA-approved conventional farming practices.

Given these deteriorating market conditions, our bodies, which spent eons evolving complex sensorial and cognitive pathways to determine whether something was good, or bad, based on appearance, taste, smell, etc., are increasingly being chemically manipulated through food science trickery. And as the food in its unadulterated state becomes more and more unappetizing, if not clearly disgusting, the slick marketing and associated nutritional disinformation becomes less and less effective on the consumer.  Enter MSG, a virtual miracle worker when it comes to turning disgusting into delicious….

Turning Yucky To Yummy With The MSG Sleight of Hand

Monosodium glutamate (MSG) is a commonly used “flavor enhancer,” and so powerfully so that (hyperbole permitting) you could spray it on roadkill and it would taste good. This omnipresent ingredient in modern mass market food takes advantage of our biologically hard-wired taste receptors, and makes it very hard to stop eating the foods “seasoned” with this ingredient. In fact, it is doubtful that without the MSG trick many of these mass market processed foods would be palatable enough to maintain their status as economically viable commodities. Here are some of its many disguises on food labels….

MSG Synonyms:

• Glutamic Acid


• Hydrolyzed protein


• Autolyzed protein


• Textured protein


• Yeast extract


• Autolyzed yeast extract


• Protein isolate


• Soy sauce


• Modified food starch


• Modified corn starch


• Calcium caseinate


• Sodlium caseinate


• Broth


• Maltodextrin


• Seasonings


• Natural flavor


• Monopotassium glutamate


• Glutamate


• Gelatin


• Hydrolyzed vegetable protein


• Hydrolyzed plant protein


• Textured protein


• Yeast food


• Yeast nutrient


• Torula yeast

 SOURCE: Indigo Earth

Technically MSG is the sodium salt of glutamic acid, a naturally occurring non-essential amino acid. Glutamic rich foods include wheat, dairy, corn, soy, seafood, etc. (Foods Highest In Glutamic Acid). The “YUMMY!” sensation that occurs immediately after ingesting a MSG (or various synonyms, e.g. hydrolyzed protein, autolyzed yeast) laced morsel the Japanese call umami (meaning: savoriness) and is considered one of five basic tastes.

The problem is that when one isolates out of a complex food a singular amino acid, and increase the concentration to unnatural proportions (and without the hundreds of checks and balances Nature provides in the context of a whole food), glutamic acid can have devastating health effects, not the least of which is the generation of an insatiable appetite for more of the very same chemical stimulating the craving — a vicious, self-amplifying cycle!

Monosodium Glutamate Causes Excitotoxicity

One of the primary adverse effects associated with excess glutamic acid is excitotoxicity, a form of neurotoxicity where neurons are stimulated to the point of cell death. Repeated excitotoxic events can result in neuronal lesions and loss of cognitive function.

While there are a number of natural substances that mitigate this type of excitotoxicity, the best choice is to reduce the consumption of glutamic acid (as well as its “twin” excitotoxic non-essential amino acid aspartic acid) rich foods, especially if there is a pre-existing neurological condition such as migraines, epilepsy or multiple sclerosis, to name but a few.  John Symes has written an excellent document on the benefits of the Glutamic and Aspartic Acid Reduced Diet (GARD Diet) here.

More Than An Excitotoxic Agent: An Endocrine Disruptor

Recently Dr. Mercola featured the connection between MSG and obesity.  While excessive food cravings caused by MSG’s taste-enhancing effects figure into this relationship, research from the US National Library of Medicine indexed on our site shows that MSG may directly cause hypothalamic lesions that result in elevated insulin, insulin resistance and leptin resistance (leptin suppresses appetite).

It is becoming clear that MSG can no longer be considered simply a “flavor enhancer” but an intrinsically harmful chemical with endocrine disruptive properties. Research we have collected shows that MSG actively contributes to metabolic syndrome, obesity, fatty liver, dysregulated blood lipids, as well as a wide range of neurological problems.

In a nutshell, monosodium glutamate (MSG) contributes to illness in two distinct ways:

1) It makes food that is bad for us taste really, really, really good, in essence compromising our health by tricking our taste buds and intuition into eating things that are intrinsically harmful, or harmful when eaten excessively.

2) It is a toxic chemical that directly damages neurological tissue through its excitotoxic properties, as well as inducing a generalized endocrine disruption throughout the body known as “metabolic syndrome,” the symptoms of which include hypertension, insulin resistance, elevated blood lipids and/or elevated blood sugar.[1] [2] [3]

---

[1] Effect of trans-fat, fructose and monosodium glutamate feeding on feline weight gain, adiposity, insulin sensitivity, adipokine and lipid profile. Br J Nutr. 2011 Mar 24:1-10. Epub 2011 Mar 24.

[2] Effect of dietary monosodium glutamate on trans fat-induced nonalcoholic fatty liver disease. J Lipid Res. 2009 Aug;50(8):1521-37. Epub 2008 Nov 11.

[3] Effects of bezafibrate in nonalcoholic steatohepatitis model mice with monosodium glutamate-induced metabolic syndrome. Eur J Pharmacol. 2011 Jul 15;662(1-3):1-8. Epub 2011 May 1.

 © February 28, 2018 GreenMedInfo LLC. This work is reproduced and distributed with the permission of GreenMedInfo LLC. Want to learn more from GreenMedInfo? Sign up for the newsletter here http://www.greenmedinfo.com/greenmed/newsletter.

The post MSG: Delicious Seasoning Or Drug And Poison? appeared first on The Sleuth Journal.

8 Things You May Not Know About Lead Poisoning

Lead is a toxic metal and lead poisoning can have really ugly consequences. Lead exposure commonly happens by way of lead based paint, the CDC has reported it as the number one cause of exposure in children. Other persons are exposed to lead dust through occupational hazards. But did you know lead exposure can cause sexual dysfunction? Or that, not long ago, multiple cases of lead poisoning resulted from contaminated flour? Here are 8 things you may not know about lead and lead poisoning.

1. Lead Exposure May Cause Schizophrenia

Researchers at Columbia University Mailman School of Public Health found that lead exposure can negatively affect the areas of the brain that are linked to schizophrenic behavior. ^[1]

2. Lead Poisoning Can Cause Erectile Dysfunction

Research conducted at Turkey’s Konya Research and Training Hospital found that chronic lead exposure was a factor responsible for increased frequency of erectile dysfunction. ^[2]

3. Lead Poisoning Can Unexpectedly Affect Pregnant Women

We all know lead poisoning is toxic and its horrible effect is multiplied when it affects pregnant women. That said, many pregnant women take extra precautions to avoid toxic metal exposure. But what if exposure happened years prior?

Lead may stay in the bones, immobilized, for decades and only mobilized when calcium needs increase during pregnancy. This leads to pregnant women and their unborn children being at a higher risk for complications from lead exposure, including anemia, high blood pressure, low birth weight, and developmental delays. This can be extremely frightening because the length in time between exposure and effect can make evaluation very difficult. ^[3]

4. Lead is Toxic to Animals

The focus on lead poisoning is not limited to humans. The Department of Biomedical Sciences at Tufts University School of Veterinary Medicine reviewed 45 years worth of literature and found 70 incidences that involved cats. Symptoms of poisoning can be less apparent in animals and researchers warned that lead toxicosis may be an under recognized problem in felines. ^[4]

5. Lead is Toxic to People Who Eat Animals

Hunting is usually done with lead ammunition. While previously dismissed as an “unlikely source of lead exposure” researchers have more recently warned that lead bullets can fragment upon impact and particles can distribute, polluting the meat. This is a potential lead exposure risk for anyone who ingests the meat of wild game that was hunted with lead ammunition. ^[5]

This has been of particular concern in Greenland. Research has shown a close relationship between how often adults consume wild birds taken during hunting and measured levels of lead in blood. The situation is severe enough that lead shot is fingered as the primary source of lead exposure for persons in Greenland. ^[6]

6. Lead has Been Found in Children’s Toys

In 2003, the Deschutes County Health Department and the Oregon Department of Human Services reported a lead poisoning incident involving a boy who had swallowed a metal trinket that was sold in a vending machine. ^[7]

Additionally, the Department of Chemistry at Ashland University in Ohio found, when evaluating paint scraped off plastic jewelry items, lead limits to be in excess of regulatory limits. Furthermore, these were on items intended for children! Just another reason to avoid plastic junk. ^[8]

7. Lead Can Significantly Contaminate Food

In 1996, a community in Chile was the subject of a massive lead poisoning incident caused by contamination of flour. Apparently, a grinding stone at the flour mill had been repaired with lead. Unfortunately, persons under six year of age at the time of exposure had a low IQ when 10 year follow up measurements were evaluated. ^[9]

8. Lead May Have Killed Beethoven

The Department of Psychiatry at the University of Ottawa conducted a toxicological analysis of Beethoven’s hair and discovered high lead levels. Lead was a common, albeit illegal, additive to wine during the 18th and 19th centuries. Researchers hypothesized Beethoven may have actually died due to a combination of liver cirrhosis, lead poisoning, and kidney failure, rather than the syphilis that is often cited. ^[10]

References (9)

1. Guilarte TR, Opler M, Pletnikov M. Is lead exposure in early life an environmental risk factor for Schizophrenia? Neurobiological connections and testable hypotheses. Neurotoxicology. 2012 Jun;33(3):560-74. doi: 10.1016/j.neuro.2011.11.008. Epub 2011 Dec 9. Review.


2. Gonulalan U, Hayırlı A, Kosan M, Ozkan O, Yılmaz H. Erectile dysfunction and depression in patients with chronic lead poisoning. Andrologia. 2012 Nov 1. doi: 10.1111/and.12029.


3. Alba A, Carleton L, Dinkel L, Ruppe R. Increased lead levels in pregnancy among immigrant women. J Midwifery Womens Health. 2012 Sep-Oct;57(5):509-14. doi: 10.1111/j.1542-2011.2012.00166.x. Epub 2012 Aug 21. Review.


4. Knight TE, Kumar MS. Lead toxicosis in cats-a review. J Feline Med Surg. 2003 Oct;5(5):249-55. Review.


5. Pain DJ, Cromie RL, Newth J, Brown MJ, Crutcher E, Hardman P, Hurst L, Mateo R, Meharg AA, Moran AC, Raab A, Taggart MA, Green RE. Potential hazard to human health from exposure to fragments of lead bullets and shot in the tissues of game animals. PLoS One. 2010 Apr 26;5(4):e10315. doi: 10.1371/journal.pone.0010315.


6. Johansen P, Pedersen HS, Asmund G, Riget F. Lead shot from hunting as a source of lead in human blood. Environ Pollut. 2006 Jul;142(1):93-7. Epub 2005 Nov 8.


7. Centers for Disease Control and Prevention (CDC). Lead poisoning from ingestion of a toy necklace–Oregon, 2003. MMWR Morb Mortal Wkly Rep. 2004 Jun 18;53(23):509-11.


8. Yost JL, Weidenhamer JD. Lead contamination of inexpensive plastic jewelry. Sci Total Environ. 2008 Apr 15;393(2-3):348-50. doi: 10.1016/j.scitotenv.2008.01.009. Epub 2008 Feb 19.


9. Coria C, Cabello A, Tassara E, López E, Rosales H, Pérez M, Zavala C, Muñoz P, Orellana G, Inostroza MI, Contreras L, Kirsten L. [Long term consequences among children exposed to lead poisoning]. Rev Med Chil. 2009 Aug;137(8):1037-44. doi: /S0034-98872009000800006. Epub 2009 Nov 4. Spanish.


10. Mai FM. Beethoven’s terminal illness and death. J R Coll Physicians Edinb. 2006 Oct;36(3):258-63.

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Toxic Metal: The Health Dangers of Tin

When most people think about tin, they imagine tin cans, tin foil, and maybe even Rin Tin Tin. Tin is a naturally occurring metal of a familiar silver color. Tin is resistant to corrosion and often used as a coating for other metals such as steel. The most common example is the use of tin to line the insides of beverage cans and food containers.Tin has other metallurgic application- when it is alloyed with copper, bronze is the result. If you’ve ever seen pewter figurines or collectibles, know that they can contain up to 90% tin. Tin is a component of solder- a regular tool for plumbing applications and electrical circuits. If you check your toothpaste label you are likely to notice the ingredient “stannous fluoride” which is a tin compound. Despite, or in light of, this frequency of exposure, concerns have been raised about the safety of tin and research has shown that tin can negatively impact human health.

Exposure to Tin

Food containers that contain tin usually have a very fine layer of lacquer applied to prevent the tin from leeching into your food or drink. Unfortunately, acidic foods may compromise that protective film. If you eat seafood, be mindful of its origin as tin has been found in seafood caught off certain costal waters. Household products like toothpaste and soap may have tin compounds added.

Manufacturing and industrial facilities are often guilty of releasing toxic metals like tin into the environment. This not only affects workers, but people who live in the vicinity too (and really, all of us). High concentrations of tin are usually found in air and soil samples in and around areas where hazardous waste is present. This can present groundwater contamination concerns.

Exposure to tin is often a byproduct of modern society. Taiwan, for example, has been changing from an agricultural society to an industrial society over the past 40 years. Due to this industrial progression, incidence of occupational neurotoxic disease has increased. Most neurotoxic diseases stem from exposure to various toxic metals, including tin, and the nervous system is especially vulnerable. ^[1]

Tin’s Negative Effects on Health

Tumor formations have been observed in the lungs of rats that inhaled dust containing tin. ^[2]

The University of Medical Sciences in Iran investigated in vitro effects of several metals, including tin, on sperm creatine kinase. Reduced sperm metabolism was observed which is believed to be a cause of infertility in men. ^[3]

Exposure to arsenic, cadmium, lead, mercury, and tin has been shown to affect the hematological (blood) system. ^[4]

Tin dust can irritate the skin and delicate tissue, particularly the eyes and respiratory system. ^[5]

Two studies out of Japan confirmed adverse effects from tin exposure to the lungs, particularly occupational lung disease. ^[6] [7]

In a study by the Department of Biotechnology and Molecular Biology at Opole University, tin was found to be extremely topic to human embryonic kidney cells. ^[8]

The nervous system is a target for a number of metals. Aluminum, arsenic, lead, and mercury are known to be incredibly neurotoxic. Lead and tin are thought to affect energy metabolism and can stall brain function by interfering with neurotransmitters. ^[9]

How Can You Reduce Your Tin Exposure?

At this time, neither the Environmental Protection Agency, the Department of Health and Human Services, or the International Agency for Research on Cancer have yet to classify tin compounds as carcinogenic to humans so regulation is not as stringent as may be appropriate. The prevalence of tin and tin compounds in industry makes it difficult to completely avoid exposure but there are a few measures you can take. With regard to diet, eat less canned food and don’t eat seafood caught from areas known to be contaminated with tin or other toxic metals. If your occupation includes safety hazards like toxic metal exposure, your risks levels are especially high.

Tests are available to determine the presence of tin compounds in the body but do not indicate where or when exposure happened. If you’re concerned about the effect of toxic metals in your body, I recommend performing a chemical and toxic metal cleanse.

References (9)

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   1. Liu CH, Huang CY, Huang CC. Occupational neurotoxic diseases in taiwan. Saf Health Work. 2012 Dec;3(4):257-67. doi: 10.5491/SHAW.2012.3.4.257. Epub 2012 Nov 30.
   
   
   2. Jiang GY, Wei SJ, Li XP, Wang LH, Mai ZD, Ge XM. [Pathological observation of lung injury in experimental animals induced by non-ferrous metal (tin) dusts]. Zhonghua Lao Dong Wei Sheng Zhi Ye Bing Za Zhi. 2012 Aug;30(8):561-6. Chinese.
   
   
   3. Ghaffari MA, Motlagh B. In vitro effect of lead, silver, tin, mercury, indium and bismuth on human sperm creatine kinase activity: a presumable mechanism for men infertility. Iran Biomed J. 2011;15(1-2):38-43.
   
   
   4. Roney N, Abadin HG, Fowler B, Pohl HR. Metal ions affecting the hematological system. Met Ions Life Sci. 2011;8:143-55.
   
   
   5. R.C.BrowneEncyclopedia of occupational health and safety col. ll/L-Z.Br J Ind Med. c.31(1);jan 1974.(1):75.
   
   
   6. Chonan T, Taguchi O, Omae K. Interstitial pulmonary disorders in indium-processing workers. Eur Respir J. 2007 Feb;29(2):317-24. Epub 2006 Oct 18.
   
   
   7. Nogami H, Shimoda T, Shoji S, Nishima S. [Pulmonary disorders in indium-processing workers]. Nihon Kokyuki Gakkai Zasshi. 2008 Jan;46(1):60-4. Japanese.
   
   
   8. Boniewska-Bernacka E, Man D, Słota R, Broda MA. Effect of tin and lead chlorotriphenyl analogues on selected living cells. J Biochem Mol Toxicol. 2011 Jul-Aug;25(4):231-7. doi: 10.1002/jbt.20380. Epub 2010 Dec 10.
   
   
   9. Clarkson TW. Metal toxicity in the central nervous system. Environ Health Perspect. 1987 Nov;75:59-64. Review.